Frequency repair, also known as electromagnetic therapy, is an independent clinical neuroscience discipline based on the three major theories of "central nervous system repairability theory", "overall frequency repair theory", and "frequency repair law" combined with induction mechanics. During screening, the device terminal emits radio resonance waves in the same frequency band as the brain waves for resonance (i.e., the interaction between physical signals and physiological signals). By detecting the frequency of ultra-low frequency biological wave signals in the human central nervous system, comparing and analyzing them with normal frequencies in the system database, the activity of organs, tissues, and cells can be determined, and the health status of the body can be evaluated. The essence is that nerve cells generate action potentials by resonance between peaks at low frequencies, and the data comes from changes in action potentials. During repair: Perform cell memory recovery training on deviated cells, and use the appropriate standard frequency emitted by the equipment to correct the deviated frequency, improve cell activity, and enhance cell function. By intervening in nerve and brain mechanical waves, the command system is restored to normal. Through brain computer interaction technology, the principle of resonance between radio resonance waves and brain biomechanical waves is used to screen, evaluate, intervene, and repair physical health status.

Nature Screening (Nonlinear Analysis) Jumping sites/3-5 cell functions Preventive Medicine: Acoustic Waves, Optical Waves, Frequency Waves, Three Wave Resonance Capture 0.1-8 HZ Cell advance/prevention/prediction Non invasive physical frequency wave quantum level

Nature What content can the system screen for? Detailed screening can be conducted for the whole body of the human body, including: 1403 organs; 5894 pathological morphological indicators; 489 common biochemical indicators; 172 microbial indicators; 16 tumor markers. Nature System Characteristics Nature The system integrates cloud services, cloud diagnosis, cloud monitoring, and cloud repair. √ Non invasive and radiation free Quick detection (detailed inspection usually within 45 minutes) √ No need to fast √ No need to draw blood √ Low cost of use √ Covering all systems and organs throughout the body Early detection of health risks and provision of intervention plans √ No harm to the body, can be checked multiple times, and the physical condition can be understood at any time

The impact of depression on the spine (1) Chronic pain and muscle tension: Muscle tension and spasms: Long term depression or anxiety can lead to sustained excitation of the sympathetic nervous system, causing chronic muscle tension throughout the body (especially in the neck, shoulders, and back), resulting in "pressure muscle pain". • Increased pain sensitivity: Patients with depression have a lower pain threshold, which may be due to central sensitization that amplifies their perception of pain. • Inflammatory response: pro-inflammatory cytokines associated with depression (such as IL-6、TNF-α） Elevated levels may exacerbate inflammatory pain in joints and muscles. (2) Indirect damage to bone health: Hormonal imbalance: Long term stress leads to an increase in cortisol (stress hormone) levels, inhibits osteoblast activity, increases the risk of osteoporosis, and indirectly affects spinal stability. • Reduced activity: Patients with depression often reduce their exercise due to decreased energy and loss of interest, leading to muscle atrophy and weakened spinal support, further exacerbating lower back pain. (3) Neuroplasticity changes: Abnormal brain spinal cord signals: Decreased neurotransmitters related to depression, such as serotonin and norepinephrine, may affect the spinal cord's ability to regulate pain signals, forming a vicious cycle of "pain depression". The impact of depression on the gastrointestinal tract (1) Dysregulation of Gut Brain Axis: • Abnormal vagus nerve function: The vagus nerve is the main channel for communication between the intestine and the brain, and depression may inhibit its function, leading to abnormal gastrointestinal motility (such as constipation or diarrhea). Disruption of gut microbiota: Depressed patients often experience a decrease in gut microbiota diversity and an increase in harmful bacteria (such as an imbalance in the proportion of Bacteroidetes/Firmicutes), leading to intestinal inflammation and metabolic abnormalities (such as a decrease in short chain fatty acids). Dual effects of serotonin (5-HT): The intestine is the site of 90% serotonin synthesis in the human body, and depression related serotonin system disorders may directly lead to intestinal motility abnormalities (such as irritable bowel syndrome, IBS）。 (2) Abnormal digestive function: Dysregulation of gastric acid secretion: Anxiety and stress stimulate gastric acid secretion through the sympathetic nervous system, increasing the risk of gastritis and gastric ulcers. • Functional gastrointestinal disease: the incidence rate of functional dyspepsia (FD) and IBS in depressed patients is significantly higher, which is manifested by abdominal pain, abdominal distention, and changes in bowel habits. (3) Immune and inflammatory response: • Damage to intestinal mucosal barrier: Chronic pressure leads to increased intestinal permeability ("intestinal leakage"), bacterial endotoxins (such as...) LPS） Entering the bloodstream, it triggers systemic low-grade inflammation, further exacerbating depressive symptoms. • Immune cell activation: pro-inflammatory cytokines (such as IL-1β、IL-6） It is transmitted to the brain through the vagus nerve, exacerbating emotional disorders and gastrointestinal discomfort.

The impact of spinal problems on depression (1) The neurobiological mechanisms of chronic pain: • Neurotransmitter imbalance: Chronic pain caused by spinal diseases such as disc herniation and arthritis can inhibit the synthesis of "pleasure neurotransmitters" such as serotonin and norepinephrine in the brain, leading to low mood. • Central sensitization: Long term pain signals lead to overactivity of pain processing areas in the spinal cord and brain, forming a "pain depression" neural circuit and reducing emotional regulation ability. • Release of inflammatory factors: Spinal degenerative diseases are often accompanied by local inflammation (such as elevated IL-6 and TNF - α), and these pro-inflammatory factors can enter the central nervous system through the blood-brain barrier, directly inhibiting hippocampal nerve regeneration and exacerbating depression. (2) Restricted activities and psychosocial impact: • Decreased physical function: Spinal problems lead to difficulty in movement, limiting daily activities such as exercise and work, causing feelings of helplessness and loss of self-worth. • Social isolation: Long term bed rest or pain may reduce social interaction and increase the risk of depression due to loneliness. • Economic pressure: The economic burden caused by reduced treatment costs and income has become an important source of psychological stress. (3) The vicious cycle of sleep disorders: • Spinal pain often leads to frequent awakenings or decreased sleep quality at night, and sleep deprivation further weakens the prefrontal cortex's ability to regulate emotions, forming a cycle of "pain insomnia depression". (1) Pathological mechanism of Gut Brain Axis Disruption of gut microbiota: Disruption of gut microbiota (such as decreased probiotics and increased pathogenic bacteria) can reduce the synthesis of short chain fatty acids (SCFAs), which are crucial for neuroprotection and anti-inflammatory effects. The reduction of microbial metabolites (such as tryptophan) directly affects the production of serotonin in the brain. • Abnormal vagus nerve signal: Intestinal inflammation or functional disorders (such as irritable bowel syndrome) transmit abnormal signals to the brain through the vagus nerve, activating emotion related brain areas such as the amygdala. • Leaky Gut and systemic inflammation: When the intestinal barrier function is impaired, bacterial endotoxins (such as LPS) enter the bloodstream, causing systemic low-grade inflammation and promoting neurodegeneration in depression related brain areas (such as the hippocampus). (2) Psychological burden of digestive symptoms • The impact of functional disorders: Recurrent abdominal pain, diarrhea, or constipation (such as IBS) can cause patients to overly focus on symptoms, leading to health anxiety and catastrophic thinking. • Nutritional absorption disorders: Long term indigestion may cause deficiencies in nutrients such as vitamin B12, iron, and magnesium, which are essential raw materials for neurotransmitter synthesis. • Shame and avoidance behavior: Avoiding social activities due to frequent toileting or dietary restrictions exacerbates feelings of loneliness and self doubt. (3) Interaction between immunity and endocrine system • Elevated cortisol: Chronic gastrointestinal problems activate the HPA axis, leading to sustained secretion of cortisol and inhibition of hippocampal neuronal regeneration. • Cytokine storm: Inflammatory cytokines such as IL-1 β and IL-6 released by intestinal inflammation affect the brain through blood circulation, reducing the function of the dopaminergic system.